Ted Bundy
Iron
- Joined
- Jan 2, 2026
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Testosterone Enanthate
Testosterone Cypionate
Testosterone Propionate
Typical cycle length
10 to 16 weeks
Support basics
Tadalafil
Omega-3 (fish oil)
Regular blood pressure monitoring
gpt miniconverts into estradiol and DHT, which is why it supports more than muscle and libido alone. Mood, cognition, bone health, and cardiovascular function all rely on those downstream hormones.
25% ain't even that bad, I use it for spelling checks and shit anyway
because there's jackshit of data behind them.Also how tf are supplements getting flagged as ai ? Be fr
Omega-3 isnt ideal but for someone not wanting to take tons of lab made shit/ expensive shit, its a great add inbecause there's jackshit of data behind them.
you just asked ai the mechanism of both sides and thought they'll counteract them
i not how it works
omega 3 is jackshit, makes 0 sense
and megadose melatonin is bad, it's not high dose = better with it, you'll just get headache and nausea afternormal dose
long release ester should've been the recommendation
and jfl at the 10-16 weeks cycle
not ideal even on test p
and you put it in the same group as test c & e
havent been online in some time and I see this masterpiece, mirin + bump
TestosteroneView attachment 26665Testosterone is the foundation because it’s what the male endocrine system is designed to work with.
Compounds
Testosterone Enanthate
Testosterone Cypionate
Testosterone Propionate
Typical cycle length
10 to 16 weeks
Support basics
Tadalafil
Omega-3 (fish oil)
Regular blood pressure monitoring
Primary metabolic pathway
Aromatase → estradiol
5α-reductase → DHT
Recovery difficulty
Generally straightforward
Structurally, it’s a C19 steroid with a 17β-hydroxyl group, which allows clean androgen-receptor binding and metabolism through pathways the body already recognises.
(Esterification doesn’t change the hormone itself; it simply slows release)
Once active, testosterone converts into estradiol and DHT, which is why it supports more than muscle and libido alone. Mood, cognition, bone health, and cardiovascular function all rely on those downstream hormones.
Mentally, this is usually the most stable class, with issues typically coming from estrogen mismanagement, poor sleep, or rising blood pressure rather than the compound itself.
View attachment 26672
Interesting study
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DHT-Derived AndrogensView attachment 26666DHT derivatives are already 5-alpha reduced, meaning they cannot convert into estrogen. This is what gives them their dry, tight appearance and makes them easier to manage in the short term.
Compounds
Anavar
Winstrol
Masteron
Typical cycle length
6 to 10 weeks
Support basics
Omega-3 (fish oil)
Ezetimibe
Low-dose tadalafil
Primary metabolic pathway
Non-aromatising
Hepatic metabolism (oral forms)
Recovery difficulty
Mild to moderate
Over time, HDL tends to drop, vascular protection declines, joints may feel drier, and neurosteroid production decreases.
Mentally, this often presents as increased focus paired with reduced emotional range or a more rigid mindset the longer these compounds are used.
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19-Nortestosterone DerivativesView attachment 2666919-nors are defined by removal of the 19th carbon methyl group, a small structural change that significantly alters receptor binding and downstream gene expression.
Compounds
Nandrolone
Trenbolone
Norethandrolone
Typical cycle length:
8 to 12 weeks
Support basics
High-dose melatonin
Tadalafil
Omega-3 (fish oil)
Primary metabolic pathway
Altered androgen-receptor binding
Progesterone-receptor interaction
CNS-active metabolites
Recovery difficulty
Difficult
These compounds are highly anabolic, but they are also more disruptive across multiple systems. They interact with progesterone receptors and produce central nervous system metabolites that behave differently than testosterone or DHT.
Trenbolone intensifies this through stronger and more persistent receptor binding.
Mentally, this class is most associated with sleep disruption, anxiety, blunted reward response, and mood instability, especially as exposure length increases.
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Oral 17α-Alkylated AndrogensView attachment 26667Orals work because a methyl or ethyl group is added at the 17α position, allowing the compound to survive first-pass liver metabolism.
Compounds
Anadrol
Dianabol
Halotestin
Typical cycle length
3 to 6 weeks
Support basics
NAC
TUDCA
Omega-3 (fish oil)
Primary metabolic pathway
17α-alkylated hepatic metabolism
Bile-flow interference
Recovery difficulty
Moderate
That same modification interferes with normal bile flow and hepatic processing. HDL drops quickly, liver enzymes rise, and cardiovascular strain builds early.
Orals act fast and feel strong, which makes them appealing, but the cost often shows up quickly in blood work.
Mentally, many users notice an initial boost followed by irritability, reduced stress tolerance, poorer sleep, and mental fatigue
Much love bhais
Hmu for any questions or 1:1 coaching
until next time
biomaxx
it's uni-deal, outdated, 16 weeks minimum, and even r/steroids wiki recommends 20 weeks for beginnersA 10-16 week is standard
yeah, the fish oil is truly not made i lab, and you're o=injecting steroids but worrying about "muh natural", sureOmega-3 isnt ideal but for someone not wanting to take tons of lab made shit/ expensive shit, its a great add in




I meant your timeline is fuckedOfc I put it in the same group and other test bases ??
jfl at this, too, it's not moving the needle for inflammation on roidsHigh dose melatonin acts as an antioxidant and anti-inflammatory


Melatonin's Impact on Antioxidative and Anti-Inflammatory Reprogramming in Homeostasis and Disease - PubMed https://share.google/v00Od2DfKDW0bTrnnit's uni-deal, outdated, 16 weeks minimum, and even r/steroids wiki recommends 20 weeks for beginners
also, if ester is slower, it's taking a long time to fully saturate aswell
10 weeks is in no way a smart idea
yeah, the fish oil is truly not made i lab, and you're o=injecting steroids but worrying about "muh natural", sure
it shouldn't even be included in the discussion, it's not doing any major contribution in the steroid protocol
jfl at this cope
"great add-in"
and my longer-ester recommendation was about the Tren part, for insomnia
test-p , mogs in ever way
I meant your timeline is fucked
10 weeks, when test c is taking like 4 weeks to fullly saturate
you're not reaching even close to the potential, just so much gains waste
If you were saying test p for 10-16, it would still be far from idea, but a lil less stupid
jfl at this, too, it's not moving the needle for inflammation on roids
there were 15 things with 150x stronger anti-inflammatory & antioxidant properties
+ megadose melatonin is far far worse than normal dose, go on, come up with the numbers/
dubt it makes any difference at mega doses, and even if it did, the sides aren't worth it when you have compounds designed for that
it's like eating sugar for IGF-1 or sum![]()
Rite like fucking high dose omega 3 is so retarded.because there's jackshit of data behind them.
you just asked ai the mechanism of both sides and thought they'll counteract them
i not how it works
omega 3 is jackshit, makes 0 sense
and megadose melatonin is bad, it's not high dose = better with it, you'll just get headache and nausea afternormal dose
long release ester should've been the recommendation
and jfl at the 10-16 weeks cycle
not ideal even on test p
and you put it in the same group as test c & e
You're an opp bruhRite like fucking high dose omega 3 is so retarded.
But high dose melatonin,works.
Megadosing omega 3 benefits?You're an opp bruh
Son I’m sorry I told you before omega 3s are literally RETARDED. I like omegas thoYou're an opp bruh
I use it for no reason lolSon I’m sorry I told you before omega 3s are literally RETARDED. I like omegas tho
Son hop off itI use it for no reason lol
Why lol it doesnt hurt and its not like im Mega dosing it.Son hop off it
You might think you just don’t want to eat omega 6 fatty acid like lenoic acid,but omega 3 is also a polyunsaturated fat that has multiple unstable bonds meaning that their oxidation is very prone. Even dosages of 1g a day of DHA and EPA cause excess oxidative stress. The only part where omega 3s are necessary is the brain,and the half life of omega 3s in brain tissue of literally 2.5 years. The thing is,natural omega 3 sources such as fish is balanced naturally with vitamin E,a cell protecter against oxidative stress and the usual amount of a serving of fish just has 200-500mg of omega 3 fatty acids which is suitable both for the adult and a developing person.Why lol it doesnt hurt and it’s not like im Mega dosing it.
Alright thanksYou might think you just don’t want to eat omega 6 fatty acid like lenoic acid,but omega 3 is also a polyunsaturated fat that has multiple unstable bonds meaning that their oxidation is very prone. Even dosages of 1g a day of DHA and EPA cause excess oxidative stress. The only part where omega 3s are necessary is the brain,and the half life of omega 3s in brain tissue of literally 2.5 years. The thing is,natural omega 3 sources such as fish is balanced naturally with vitamin E,a cell protecter against oxidative stress and the usual amount of a serving of fish just has 200-500mg of omega 3 fatty acids which is suitable both for the adult and a developing person.
Bc its assMirin thread but i wanted to ask how come Undecanoate isn't that commonly used


