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Discussion Let’s talk about bonesmashing (4 Viewers)

Discussion Let’s talk about bonesmashing

Dexter

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Bone homeostasis significantly limits the magnitude of mechanotransduction changes. Without exogenous growth factors, the effects are generally modest and slow, although maybe significant in a long term.
I'll make a thread about this.
looking forward to it
 

FoidSlayer

we will all ascend
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I didn't FUCKING say you were FROM there, I'm saying there is a war there and if you're so manly you could enlist. Besides it was a joke
Yh even if i had the balls to fight in iran
I still wouldn’t go because its simply not worth it dying for a country that doesn’t give a shit about u but alr
 

dior

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birthdefect

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me and surgerymaxx were actually discussing this yesterday, bone smashing works but you only gain scar tissue, not real "bone", and also for it to "work" you would need to take some compound that super charges collagen synthesis, even if it theoretically works, fillers are always more convenient imo.
probably the biggest trvth nvke, although its def bone
maybe my chin skin is thin enough to where i can damage the bone easily, i literally have no meat on it and a completely flat pogonion
after a certain point bonesmashing requires such force and its such a strong negative feedback loop i just cant be bothered to do it
although i will hopefully prove my chin bumps by getting my skull scanned soon
 

birthdefect

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It works, and it works through the sclerostin and Wnt/β-catenin axis. Subperiosteal hematomas is a retarded theory; you won’t achieve consistent blood pooling, and even if you do it won’t lead to aesthetic results.
its light work, do it with a smaller object for smaller areas and profit
the blood pooling isnt random, it only occurs where you hit then expands outwards into a small oblate spheroid shape
elaborate on what you mean by it working through "the sclerostin and Wnt/β-catenin axis"
bone growth occurs from many different growth pathways and downregulators, sost isnt even the only one to manage you gotta inhibit dkk1 for the best results tbh
 

birthdefect

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Bone homeostasis significantly limits the magnitude of mechanotransduction changes. Without exogenous growth factors, the effects are generally modest and slow, although maybe significant in a long term.
I'll make a thread about this.
tag me in it as well
 

Synapzyzz

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probably the biggest trvth nvke, although its def bone
maybe my chin skin is thin enough to where i can damage the bone easily, i literally have no meat on it and a completely flat pogonion
after a certain point bonesmashing requires such force and its such a strong negative feedback loop i just cant be bothered to do it
although i will hopefully prove my chin bumps by getting my skull scanned soon
Most people won’t bother to do proper research and actually understand how can it “theoretically” work, for most people doing a conservative amount of filler it’s the best option, way more convenient for people who are not willing to have to deal with the sides of Winstrol or any other steroids that supercharges collagen synthesis, also learning to DIY fillers is not that hard, orka, dior, veridic and other users have managed to do this without any issues, and the migration risk is low when the supraperiosteal injection is done properly, also HA fillers are relatively cheap, 60$ plus the needles and syringes, the numbing cream and if u fucked yourself the hyaluronidase, ofc fillers are not a long term solution, but you could probably get some good results injecting and after 12 months dissolving and repeating, till you manage to get the money for implants ofc.
 

birthdefect

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Most people won’t bother to do proper research and actually understand how can it “theoretically” work, for most people doing a conservative amount of filler it’s the best option, way more convenient for people who are not willing to have to deal with the sides of Winstrol or any other steroids that supercharges collagen synthesis, also learning to DIY fillers is not that hard, orka, dior, veridic and other users have managed to do this without any issues, and the migration risk is low when the supraperiosteal injection is done properly, also HA fillers are relatively cheap, 60$ plus the needles and syringes, the numbing cream and if u fucked yourself the hyaluronidase, ofc fillers are not a long term solution, but you could probably get some good results injecting and after 12 months dissolving and repeating, till you manage to get the money for implants ofc.
i didnt need anything to get the growth but i do agree
the bone strengthens, and constantly hitting it is just not a good experience
 

Tabula Rasa

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its light work, do it with a smaller object for smaller areas and profit
the blood pooling isnt random, it only occurs where you hit then expands outwards into a small oblate spheroid shape
1- You cannot prove that your bonesmashing technique produces subperiosteal hematomas, and given that this is a rare condition, it’s naive to assume that a self-inflicted trauma would produce it, much less produce it consistently.
2- Most subperiosteal hematomas do not ossify, and many factors are required for that process to occur.
3- Subperiosteal hematomas, tend to expand; to achieve aesthetic augmentation you would need consistent blood pooling across the entire bone you want to augmentate, which you are not going to achieve, let alone trigger a process of ossification.
In other words, it's playing with small possibilities within other small possibilities, within infinitesimal possibilities. A doll of impossibility, which makes its consistency impossible.
Russian Dolls Babushka GIF by Barbara Pozzi
elaborate on what you mean by it working through "the sclerostin and Wnt/β-catenin axis"
bone growth occurs from many different growth pathways and downregulators, sost isnt even the only one to manage you gotta inhibit dkk1 for the best results tbh
I did not say that it is the only mechanism of bone growth, but rather the one by which mechanotransduction works.

https://pmc.ncbi.nlm.nih.gov/articles/PMC5987014/

Sclerostin, an emerging therapeutic target for treating osteoporosis and osteoporotic fracture: A general review

Sclerostin inhibits bone formation through inhibiting the Wnt/β-catenin signalling​

The expression of sclerostin is upregulated in tibia in a hindlimb unloading model (tail suspension) in mice and downregulated in ulna in loading model in both rat and mice​



TL;dnr:
Sclerostin is a regulator of this mechanism, inhibiting Wnt/β-catenin to prevent bone growth. This also suppresses osteoblastogenesis, the process by which mesenchymal stem cellsare committed toward osteoblasts, essential bone building cells.


 

Kramatorsk1131

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0/10 cope, while Orka Orka Veridic Veridic and dior dior are ascending you are coping like an absolute retard about "muh fillers for women".
keep glazing ur bfs
 

Kramatorsk1131

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bonesmashing works but you need to use another mans penis to hit your bones, otherwise it wont work, dont ask me why tho
 

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