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Mogs Soteraxfin (GTFIH) (1 Viewer)

Mogs Soteraxfin (GTFIH)
  • Thread starter Biomaxx
  • Start date

Biomaxx

Xi Jinping's pharmacist
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  • #1
I think ive came across a god made compound that I probably shoudnt be writing about

SRX-9147 (Soteraxifin): First-In-Class Osteo-Anabolic Mitogen

soreraxfin has just had its first published animal trail (rats)

Soteraxifin was administered 100mcg/kg to teenage rats and showed (imo) incredible results

Results showed- insane femur thickening and lengthening, large increase in bmd, near complete halt in chondrocytes differentiation (plate fusure) and slight myostatin inhibition (more lean muscle)

This compound was seen to work through 4 different main pathway

1.FGFR3 Pathway Inhibition
Unlike expensive antibody approaches, Soteraxfin is a small molecule FGFR3 inhibitor that penetrates the growth plate. Reduces constitutive STAT1 phosphorylation, allowing chondrocytes to stay proliferative longer. No impact on FGFR1/2/4 at therapeutic doses.

2. Wnt/β-Catenin Potentiation
Standard Wnt agonists hit LRP5/6 and wreck your gut. Soteraxfin amplifies endogenous Wnt signaling downstream at the Dvl/Axin level. Result: significant BMD accretion without the GI sides that killed other candidates in this class.

3. Myostatin Latency Maintenance
Binds the myostatin pro-domain with high affinity, preventing proteolytic activation. Serum active myostatin dropped 94% in the 100mcg cohort. Lean mass up 4.2kg in 8 weeks without training protocol changes.

4. CNP/NPR-B Signaling Amplification
Positive allosteric modulator at the NPR-B receptor. Endogenous CNP stays active 3-4x longer in growth plate tissue. cGMP elevation localized to bone - no systemic hypotension or vascular effects seen.

Results over a month-
Screenshot_20260401_143205_Kimi.jpg

These results are Unlike anything tested

Side Effects
Reportedly near to none
Mild headaches and early usage stool softening

⚠️⚠️⚠️⚠️ OPEN SPOILER​
April fools, silly mf 😘

birthdefect birthdefect @Dexter Dexter syna Synapse Fr4mel3t Fr4mel3t carbon carbon tmpll tmpll FoidSlayer FoidSlayer XvideosDemon XvideosDemon
 

Biomaxx

Xi Jinping's pharmacist
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  • #2
Jfl why do I even post bruh 😭 so bored
 

Biomaxx

Xi Jinping's pharmacist
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  • #3
Make sure u open the spoiler
 
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  • #4
I think ive came across a god made compound that I probably shoudnt be writing about

SRX-9147 (Soteraxifin): First-In-Class Osteo-Anabolic Mitogen

soreraxfin has just had its first published animal trail (rats)

Soteraxifin was administered 100mcg/kg to teenage rats and showed (imo) incredible results

Results showed- insane femur thickening and lengthening, large increase in bmd, near complete halt in chondrocytes differentiation (plate fusure) and slight myostatin inhibition (more lean muscle)

This compound was seen to work through 4 different main pathway

1.FGFR3 Pathway Inhibition
Unlike expensive antibody approaches, Soteraxfin is a small molecule FGFR3 inhibitor that penetrates the growth plate. Reduces constitutive STAT1 phosphorylation, allowing chondrocytes to stay proliferative longer. No impact on FGFR1/2/4 at therapeutic doses.

2. Wnt/β-Catenin Potentiation
Standard Wnt agonists hit LRP5/6 and wreck your gut. Soteraxfin amplifies endogenous Wnt signaling downstream at the Dvl/Axin level. Result: significant BMD accretion without the GI sides that killed other candidates in this class.

3. Myostatin Latency Maintenance
Binds the myostatin pro-domain with high affinity, preventing proteolytic activation. Serum active myostatin dropped 94% in the 100mcg cohort. Lean mass up 4.2kg in 8 weeks without training protocol changes.

4. CNP/NPR-B Signaling Amplification
Positive allosteric modulator at the NPR-B receptor. Endogenous CNP stays active 3-4x longer in growth plate tissue. cGMP elevation localized to bone - no systemic hypotension or vascular effects seen.

Results over a month-
View attachment 41457
These results are Unlike anything tested

Side Effects
Reportedly near to none
Mild headaches and early usage stool softening

⚠️⚠️⚠️⚠️ OPEN SPOILER​
April fools, silly mf 😘

birthdefect birthdefect @Dexter Dexter syna Synapse Fr4mel3t Fr4mel3t carbon carbon tmpll tmpll FoidSlayer FoidSlayer XvideosDemon XvideosDemon
First high effort April fools thread I've seen, peak
 

sensitive sapphire

autosexual · From Church of Preet
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  • #5
mirin(im hurt i wasent tagged)
 

Biomaxx

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  • #6

bob08674

Bye guys
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  • #7
Very nice thread deffinetly read and understood everything
 

Hauptmann

cl by 2027
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  • #8
:no:
 
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  • #9
Can I combine It with maasai jumps or will I grow too much?
 

Biomaxx

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  • #10

FoidSlayer

we will all ascend
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  • #11
Nice spoiler
 
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  • #12
I think ive came across a god made compound that I probably shoudnt be writing about

SRX-9147 (Soteraxifin): First-In-Class Osteo-Anabolic Mitogen

soreraxfin has just had its first published animal trail (rats)

Soteraxifin was administered 100mcg/kg to teenage rats and showed (imo) incredible results

Results showed- insane femur thickening and lengthening, large increase in bmd, near complete halt in chondrocytes differentiation (plate fusure) and slight myostatin inhibition (more lean muscle)

This compound was seen to work through 4 different main pathway

1.FGFR3 Pathway Inhibition
Unlike expensive antibody approaches, Soteraxfin is a small molecule FGFR3 inhibitor that penetrates the growth plate. Reduces constitutive STAT1 phosphorylation, allowing chondrocytes to stay proliferative longer. No impact on FGFR1/2/4 at therapeutic doses.

2. Wnt/β-Catenin Potentiation
Standard Wnt agonists hit LRP5/6 and wreck your gut. Soteraxfin amplifies endogenous Wnt signaling downstream at the Dvl/Axin level. Result: significant BMD accretion without the GI sides that killed other candidates in this class.

3. Myostatin Latency Maintenance
Binds the myostatin pro-domain with high affinity, preventing proteolytic activation. Serum active myostatin dropped 94% in the 100mcg cohort. Lean mass up 4.2kg in 8 weeks without training protocol changes.

4. CNP/NPR-B Signaling Amplification
Positive allosteric modulator at the NPR-B receptor. Endogenous CNP stays active 3-4x longer in growth plate tissue. cGMP elevation localized to bone - no systemic hypotension or vascular effects seen.

Results over a month-
View attachment 41457
These results are Unlike anything tested

Side Effects
Reportedly near to none
Mild headaches and early usage stool softening

⚠️⚠️⚠️⚠️ OPEN SPOILER​
April fools, silly mf 😘

birthdefect birthdefect @Dexter Dexter syna Synapse Fr4mel3t Fr4mel3t carbon carbon tmpll tmpll FoidSlayer FoidSlayer XvideosDemon XvideosDemon
How bored is bro 🤣
 

nineteen

ㅤㅤㅤㅤㅤㅤㅤㅤㅤㅤㅤㅤ
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  • #13
I think ive came across a god made compound that I probably shoudnt be writing about

SRX-9147 (Soteraxifin): First-In-Class Osteo-Anabolic Mitogen

soreraxfin has just had its first published animal trail (rats)

Soteraxifin was administered 100mcg/kg to teenage rats and showed (imo) incredible results

Results showed- insane femur thickening and lengthening, large increase in bmd, near complete halt in chondrocytes differentiation (plate fusure) and slight myostatin inhibition (more lean muscle)

This compound was seen to work through 4 different main pathway

1.FGFR3 Pathway Inhibition
Unlike expensive antibody approaches, Soteraxfin is a small molecule FGFR3 inhibitor that penetrates the growth plate. Reduces constitutive STAT1 phosphorylation, allowing chondrocytes to stay proliferative longer. No impact on FGFR1/2/4 at therapeutic doses.

2. Wnt/β-Catenin Potentiation
Standard Wnt agonists hit LRP5/6 and wreck your gut. Soteraxfin amplifies endogenous Wnt signaling downstream at the Dvl/Axin level. Result: significant BMD accretion without the GI sides that killed other candidates in this class.

3. Myostatin Latency Maintenance
Binds the myostatin pro-domain with high affinity, preventing proteolytic activation. Serum active myostatin dropped 94% in the 100mcg cohort. Lean mass up 4.2kg in 8 weeks without training protocol changes.

4. CNP/NPR-B Signaling Amplification
Positive allosteric modulator at the NPR-B receptor. Endogenous CNP stays active 3-4x longer in growth plate tissue. cGMP elevation localized to bone - no systemic hypotension or vascular effects seen.

Results over a month-
View attachment 41457
These results are Unlike anything tested

Side Effects
Reportedly near to none
Mild headaches and early usage stool softening

⚠️⚠️⚠️⚠️ OPEN SPOILER​
April fools, silly mf 😘

birthdefect birthdefect @Dexter Dexter syna Synapse Fr4mel3t Fr4mel3t carbon carbon tmpll tmpll FoidSlayer FoidSlayer XvideosDemon XvideosDemon
wow

ok
 

khanlite

Muslim
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  • #14
When Methyltrienolone thread
 

MedSlayer

Former Subhuman
Joined
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2,009
  • #15
I think ive came across a god made compound that I probably shoudnt be writing about

SRX-9147 (Soteraxifin): First-In-Class Osteo-Anabolic Mitogen

soreraxfin has just had its first published animal trail (rats)

Soteraxifin was administered 100mcg/kg to teenage rats and showed (imo) incredible results

Results showed- insane femur thickening and lengthening, large increase in bmd, near complete halt in chondrocytes differentiation (plate fusure) and slight myostatin inhibition (more lean muscle)

This compound was seen to work through 4 different main pathway

1.FGFR3 Pathway Inhibition
Unlike expensive antibody approaches, Soteraxfin is a small molecule FGFR3 inhibitor that penetrates the growth plate. Reduces constitutive STAT1 phosphorylation, allowing chondrocytes to stay proliferative longer. No impact on FGFR1/2/4 at therapeutic doses.

2. Wnt/β-Catenin Potentiation
Standard Wnt agonists hit LRP5/6 and wreck your gut. Soteraxfin amplifies endogenous Wnt signaling downstream at the Dvl/Axin level. Result: significant BMD accretion without the GI sides that killed other candidates in this class.

3. Myostatin Latency Maintenance
Binds the myostatin pro-domain with high affinity, preventing proteolytic activation. Serum active myostatin dropped 94% in the 100mcg cohort. Lean mass up 4.2kg in 8 weeks without training protocol changes.

4. CNP/NPR-B Signaling Amplification
Positive allosteric modulator at the NPR-B receptor. Endogenous CNP stays active 3-4x longer in growth plate tissue. cGMP elevation localized to bone - no systemic hypotension or vascular effects seen.

Results over a month-
View attachment 41457
These results are Unlike anything tested

Side Effects
Reportedly near to none
Mild headaches and early usage stool softening

⚠️⚠️⚠️⚠️ OPEN SPOILER​
April fools, silly mf 😘

birthdefect birthdefect @Dexter Dexter syna Synapse Fr4mel3t Fr4mel3t carbon carbon tmpll tmpll FoidSlayer FoidSlayer XvideosDemon XvideosDemon
Water
 

syna

Moderator
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  • #16
mirin the high effort it took to create this stuff lmfao.
 

birthdefect

pray to the purple powder
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  • #17
I think ive came across a god made compound that I probably shoudnt be writing about

SRX-9147 (Soteraxifin): First-In-Class Osteo-Anabolic Mitogen

soreraxfin has just had its first published animal trail (rats)

Soteraxifin was administered 100mcg/kg to teenage rats and showed (imo) incredible results

Results showed- insane femur thickening and lengthening, large increase in bmd, near complete halt in chondrocytes differentiation (plate fusure) and slight myostatin inhibition (more lean muscle)

This compound was seen to work through 4 different main pathway

1.FGFR3 Pathway Inhibition
Unlike expensive antibody approaches, Soteraxfin is a small molecule FGFR3 inhibitor that penetrates the growth plate. Reduces constitutive STAT1 phosphorylation, allowing chondrocytes to stay proliferative longer. No impact on FGFR1/2/4 at therapeutic doses.

2. Wnt/β-Catenin Potentiation
Standard Wnt agonists hit LRP5/6 and wreck your gut. Soteraxfin amplifies endogenous Wnt signaling downstream at the Dvl/Axin level. Result: significant BMD accretion without the GI sides that killed other candidates in this class.

3. Myostatin Latency Maintenance
Binds the myostatin pro-domain with high affinity, preventing proteolytic activation. Serum active myostatin dropped 94% in the 100mcg cohort. Lean mass up 4.2kg in 8 weeks without training protocol changes.

4. CNP/NPR-B Signaling Amplification
Positive allosteric modulator at the NPR-B receptor. Endogenous CNP stays active 3-4x longer in growth plate tissue. cGMP elevation localized to bone - no systemic hypotension or vascular effects seen.

Results over a month-
View attachment 41457
These results are Unlike anything tested

Side Effects
Reportedly near to none
Mild headaches and early usage stool softening

⚠️⚠️⚠️⚠️ OPEN SPOILER​
April fools, silly mf 😘

birthdefect birthdefect @Dexter Dexter syna Synapse Fr4mel3t Fr4mel3t carbon carbon tmpll tmpll FoidSlayer FoidSlayer XvideosDemon XvideosDemon
NIGGA YOU GOT ME EXCITED
i was reading through that getting more and more shocked
 

Biomaxx

Xi Jinping's pharmacist
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  • #18

khanlite

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  • #19

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